Extract Reduces Acetaminophen Induced Liver Damage

<p class=ql-align-justify>Extract Reduces Acetaminophen Induced Liver Damage by P.K. Senthil Ganesh is a comprehensive book that explores the potential of herbal medicine and natural products as a hepatoprotective agent against acetaminophen-induced liver damage. The book highlights the importance of hepatoprotective agents that can reduce liver damage caused by the overuse of acetaminophen a common painkiller medication.</p><p class=ql-align-justify><br></p><p class=ql-align-justify>The book delves into the various mechanisms by which the herbal extract reduces acetaminophen-induced liver damage such as antioxidant and anti-inflammatory properties. The author explains how the extract can prevent apoptosis and necrosis of hepatocytes two major factors in acetaminophen-induced liver damage. Additionally the book explores how the extract modulates cytochrome P450 and glutathione levels which play a vital role in the detoxification process.</p><p class=ql-align-justify><br></p><p class=ql-align-justify>The book also provides a detailed overview of the role of free radicals oxidative stress cytokines chemokines and inflammation in acetaminophen-induced hepatic injury and hepatotoxicity. It further explains how the extract promotes hepatocyte regeneration and growth factor production while inhibiting proinflammatory factors such as tumor necrosis factor and interleukin.</p><p class=ql-align-justify><br></p><p class=ql-align-justify>The book also discusses the role of various immune cells such as macrophages Kupffer cells neutrophils and lymphocytes in acetaminophen-induced liver damage and how the extract can modulate their activity to promote hepatoprotective effects.</p><p class=ql-align-justify><br></p><p class=ql-align-justify>Overall Extract Reduces Acetaminophen Induced Liver Damage by P.K. Senthil Ganesh provides a comprehensive overview of the medicinal plants phytochemicals and flavonoids that can potentially reduce liver damage caused by acetaminophen-induced hepatotoxicity making it a valuable resource for researchers and clinicians interested in hepatoprotective agents.</p><p><br></p>